
Dr. Rachita Sumbria
- Education:
- Institute of Chemical Technology, Bachelor of Technology
Texas Tech University, Ph.D.
Biography
Dr. Sumbria joined Chapman University School of Pharmacy as an Associate Professor in February of 2021. Dr. Sumbria received her PhD in Pharmaceutical Sciences from the Texas Tech University Health Sciences Center (TTUHSC) in 2010. At TTUHSC, her research centered on the development of intracerebral microdialysis as an experimental tool to study changes in the blood-brain barriers (BBB) permeability in ischemic stroke.
Dr. Sumbria completed her postdoctoral training at the Department of Medicine, blood-brain barrier laboratory at the University of California, Los Angeles and the Department of Neurology at the University of California, Irvine from 2011-2014. During her postdoctoral research at UCLA, Dr. Sumbria focused on the problem BBB bottleneck in CNS drug delivery. Particularly, her research focused on the use of receptor-mediated transcytosis for non-invasive delivery of biologics and other large molecular weight therapeutics across the BBB, and studying their pharmacologic effects in different experimental models of CNS disorders. At UCI, the focus of her research was studying the role of cerebral microbleeds in cerebrovascular diseases like stroke. In 2014, she joined the faculty at the School of Pharmacy and Health Sciences at the Keck Graduate Institute as an Assistant Professor and was promoted to Associate Professor in 2019. During her academic career, Dr. Sumbria has been extensively involved in research that has been funded by federal and foundation grants, teaching and mentoring laboratory research personnel and graduate students.
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Recent Creative, Scholarly Work and Publications
- Feinberg PA, Becker SC, Chung L, Ferrari L, Stellwagen D, Anaclet C, Durán-Laforet V, Faust TE, Sumbria RK, Schafer DP*. Elevated TNF-a leads to neural circuit instability in the absence of Interferon Regulatory Factor 8. J Neurosci. 2022 Jul 1:JN-RM-0601-22. doi: 10.1523/JNEUROSCI.0601-22.2022.